Causes and Effects of the Persian Gulf War Essay -- Operation Desert S

Causes and Effects of the Persian Gulf War The Persian Gulf War, often referred to as Operation Desert Storm, was perhaps one of the most successful war campaigns in the history of warfare. Saddam Hussein, leader of Iraq, invaded Kuwait in 1990. In 1991, after weeks of air strikes, US ground forces entered Iraq and Kuwait and eliminated Iraqi presence in 60 hours. Why Would Iraq invade Kuwait? Kuwait supplies much of the world’s oil supplies, and when Hussein invaded Kuwait, he controlled 24% of the world’s oil supplies (O’Hara). Though this is a good reason, it is not the only one. Iraq’s real excuse for annexing Kuwait was that he believed that Kuwait was producing more oil than it was supposed to, taking out of Iraq’s profits. Also, Iraq was $80 billion in debt to Kuwait, and Iraq thought that the debt should be forgiven (Brown). After Iraq attacked Kuwait, the United States and other countries feared that Saudi Arabia would be next and that the world’s oil supply was in jeopardy. This was the spark that leads to the Persian Gulf War. There was also controversy with Iraq even before the invasion of Kuwait. Iraq was suspected of developing chemical weapons. The United Nations had often asked to check their weapons supplies to prevent Iraq from gaining great military power. When Iraq refused to let the UN inspectors into the country, suspicion was raised. One final thing that began the Persian Gulf War was Iraq’s development of the SCUD Missile. Though n...

Ketamine Pain Mechanism

Pain is communicated from the brain to other parts of the body by the CNS (Central Nervous System) and nerve endings. (Mayer, Mao, Holt, Price, 7731-7736) The ligand-gated ion channels, also referred to as LGICs, or ionotropic receptors, are a group of intrinsic transmembrane ion channels that are opened in response to binding of a chemical messenger. (Collingridge, Singer, 290-296) (Dickenson, 307-309) (Dickenson, Chapman, Green, 633-638)The ion channel is regulated by a neurotransmitter ligand that is very selective to one or more ions like potassium, sodium, calcium, and chloride. (Kandel, Schwartz, Jessell, 178-180)   Such receptors located at synapses converting the chemical signal to an electric signal in the post-synaptic cell. (Connolly, Wafford, 529-534)   The NMDA receptor (N-methyl-D-aspartate) is such an ionotropic receptor for glutamate. (Dingledine, Borges, Bowie, Traynelis, 7-61) (Lodge, Johnson, 81-86) (Meller, 435-436)   By X-ray crystallography, the NMDA recep tors have an heterodimer subunits, which are involved in the binding of agonists and antagonists like Ketamine. (Hirota, Lambert, 441-444)This channel complex contributes to excitatory synaptic transmission at sites throughout the brain and the spinal cord, and is modulated by a number of endogenous and exogenous compounds. (Rabben, Skljelbred, Oye, 1060-1066)   NMDA receptors play a key role in a wide range of physiologic and pathologic processes. (Hoffman, Coppejans, Vercauteren, Adriemsen, 240-242) (Klepstadt, Maurset, Moberg, Oye, 513-518) (Coderre, Katz, Vaccarino, Melzack, 259-285) Ketamine is primarily a non-competitive antagonist, which opens in response to binding of glutamate. This NMDA receptor mediates the reduction of pain effects of ketamine at low doses. (Lofwall, Griffiths, Mintzer, 439-449)Evidence for this is reinforced by the fact that naxolone, an opioid antagonist, does not reverse the analgesia. Studies also seem to indicate that ketamine is ‘use depend ent' meaning it only initiates its blocking action once a glutamate binds to the NMDA receptor. (Sorensen, Bengtsson, Ahlner, Henriksson, Ekselius et al., 1615-1621)   At high level doses, ketamine has also been found to bind to opioid mu receptors and sigma receptors. Thus, loss of consciousness that occurs may be partially due to binding at the opioid mu and sigma receptors. (Lonnqvist, Norton, 617-621)(Menigaux, Fletcher, Dupont, Guignard, Guirimand, et al. 129-135) (Koppert, Sittl, Scheuber, Alsheimer, Schmelz, 152-159) (Bushell, Endoh, Simen, Ren, Bindokas, 55-64)Works CitedMayer DJ, Mao J, Holt J, Price DD. Cellular Mechanisms of Neuropathic Pain, Morphin Tolerance, and their Interactions. Proc. Natl Acac. Sci. USA. 1999, 96(14): 7731-7736.Collingridge G, Singer W. Excitatory Amino Acid Receptors and Synaptic Plasticity. Trends Pharmacol Sci. 1990 11: 290-296.Dickenson AH. A cure for wind-up: NMDA receptor antagonists as potential analgesics. Trends Pharmacol Sci 1990 11: 30 7-309Dickenson AH, Chapman V and Green GM. The pharmacology of excitatory and inhibitory amino acid-mediated events in the transmission and modulation of pain in the spinal cord. Gen Pharmacol 1997 28: 633-638Kandel ER, Schwartz JH, Jessell TM. Principles of Neural Science, 4th ed. McGraw-Hill: New York, (2000), pp.178-180Connolly CN, Wafford KA. The Cys-Loop Superfamily of Ligand-Gated Ion Channels – the Impact of Receptor Structure on Function. Biochemical Society Transactions (2004) Vol. 32: 529-534.Dingledine R, Borges K, Bowie D, Taynelis SF. The Glutamate Receptors Ion Channels. Pharmacology Reviews, 1999 51(1): 7-61Lodge D and Johnson KM. Non-Competitive Excitatory Amino Acid Antagonists. Trends Pharmacol Sci 1990 11: 81-86Meller ST. Ketamine: Relief from Chronic Pain through Actions at the NMDA Receptor? Pain   1996 68: 435-436Hirota K, Lambert DG. Ketamine: Its Mechanism (s) of Action and its Unusual Clinical Uses. Br. J. Anesth. 1996, 77(4):441-444.Rabben T, Skjel bred P, Oye I. Prolonged Analgesic Effects of Ketamine, an N-Methyl-D-Aspartate Receptor Inhibitor, in Patients with Chronic Pain. The Journal of Pharmacology and Experimental Pharmaceutics. 1999, 289(2):1060-1066.Hoffmann V, Coppejans H, Vercauteren M and Adriaemsen H Successful Treatment of Postherpetic Neuralgia with Oral Ketamine. 1994 Clin J Pain 10: 240-242Klepstad P, Maurset A, Moberg ER and Oye I Evidence for a Role for NMDA Receptors in Pain Perception. Eur J Pharmacol   1990 187: 513-518Coderre TJ, Katz J, Vaccarino AL and Melzack R.   Contribution of Central Neuroplasticity to Pathological Pain: A Review of Clinical and Experimental Evidence. 1993 Pain 52: 259-285.Lofwall MR, Griffiths RR, Mintzer MZ. Cognitive and Subjective Acute Dose Effects of Intramuscular Ketamine in Healthy Adults. Ex. Clin. Psychopharmacol. (2006), 14(4):439-449Sorensen J, Bengtsson A, Ahlner J, Henriksson KG, Ekselius L and Bengtsson M.   Fibromyalgia. Are there different mechanisms in the processing of pain? A double Blind Crossover Comparison of analgesic Drugs. 1997 J Rheumatol 24: 1615-1621Lonnqvist PA, Norton NS. Pediatric Day-Case Anesthesia and Pain Control.   Curr. Opin. Anaest. (2006), 19(6): 617-621.Menigaux C, Fletcher D, Dupont X, Guignard B, Guirimand F, Chauvin M. The Benefits of Intraoperative Small-Dose Ketamine on Postoperative Pain after Anterior Cruciate Ligament Repair. Anesth. Analg. 2000 90(1): 129-135Koppert W, Sittl R, Scheuber K,Alsheimer M, Schmeltz M, Schuttler J. Differential Modulation of Remifentanil-Induced Analgesia and Post-Infusion Hyperalgesia by S-Ketamine and Clonidine in Humans. Anesthesiology. 2003, 99(1): 152-159.Bushell T, Endoh T, Simen AA, Ren D, Bindokas VP, Miller RJ. Molecular Components of Tolerance to Opiates In Single Hippocampal Neurons. Mol. Pharmacol. 2002, 61(1): 55-64.

Rooms Division Operation Management - 21324 Words

| Rooms Division operation Management | Higher National Diploma In Hospitality Management | Bheemuck Chuvinee Neha amp; Emile Natacha Sandrine 11/19/2012 | Acknowledgement We would take this opportunity to first of all, express our words of gratitude to Mr Armooghum, who has been present for our queries. He has helped us a lot in solving confusions. The lectures have been helpful for the assignment, which have been one of the motivating factors. We would also like to thank the organizations or rather the members of the staff who have enormously contributed by filling the questionnaires and giving their personal views and information on the topic. Thank you Contents Acknowledgement 2 Table of Figures 7 Introduction 8†¦show more content†¦This is why it is important like every staff in fact, to have a perfect grooming and a good personality. 1.1.2 Accomodation * Maintenance service. In the hotels selected, the maintenance service is the same. The maintenance service forms part of the housekeeping service, they are in charge of every technical issues such as electricity power cut, no availability of hot water, and others. * Working procedures All hotels selected has the concept of working procedures. But which are different from each other. But the working procedures which can be defined also by Standard of procedures is an important concept having the same objectives. It is applied for standardization and also for effectiveness. As per WawickShire College (2012), Standard Operating Procedure is a ‘’Written procedure prescribed for repetitive use as a practice, in accordance with agreed upon specifications aimed at obtaining a desired outcome.’’ It is important because it helps to have a better standard of the work, and also the same result required. If the SOP’s did not exist, there would be a variation of results, which will be not uniformed. The standard Operating procedures also help to have a better effectiveness of using materials, which will help to have a better lifespan of the materials. It also helps the staff to use the materials in a safe way thus ensuring the healthShow MoreRelatedRoom divisions Operations Management5493 Words   |  22 Pagesï » ¿ Rooms Division Operations Management Table of Contents Executive Summary This case study explores the importance of Room Division service in hospitality industry. The task 1 discusses the different services provided by the rooms division in different circumstances. The impact of different operational issues in managing the front of house area is evaluated in the second task. 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